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Rvations made and the hypotheses stated earlier indicate that in the absence of SP-A there is a loss of regulatory control to appropriately modulate expression of DEF proteins in response to infection. Consistent with this postulate, a recent study with SP-A-/mice demonstrated that in response to low level intrapharyngeal LPS treatment (0.5 ng), significantly higher levels of MIP-2 were observed c
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Stimulated M2 polarization. Moreover, human explant mammary gland cultures treated with rSFRP1 show a marked reduction in the human M2 marker, CD163. CD163 is a monocyte/macrophage-restricted scavenge receptor [47] and the mechanism by which rSFRP1 reduces CD163 expression may be due in part to repression of Wnt signaling because Bergenfelz et al. show that breast cancer CD163+ TAMs correlate with
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Stimulated M2 polarization. Moreover, human explant mammary gland cultures treated with rSFRP1 show a marked reduction in the human M2 marker, CD163. CD163 is a monocyte/macrophage-restricted scavenge receptor [47] and the mechanism by which rSFRP1 reduces CD163 expression may be due in part to repression of Wnt signaling because Bergenfelz et al. show that breast cancer CD163+ TAMs correlate with
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Noted in atypical breast lesions [49, 50]. Our studies suggest that loss of SFRP1 in epithelial cells can enhance TGF- mediated EGR2 expression and affect TGF- induced M2 polarization of macrophages. As M2 macrophages secrete growth factors, such as Wnt ligands and EGF, this could contribute to tumor progression through a feed forward cross talk between the epithelium and the immune system. Additi
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If you have any questions or remarks about the operation of the portal or would like to know the details of potential cooperation, we invite you to contact us:editorial officeEditorial address:Comparic.plul. Bodzentyńska 32/40 m1125-308 Kielcee-mail: redakcja@comparic.pltel. +48 731 244 014Editor-in-Chief - M
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