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Thus, it was reasonable the system of inhibition of big WT1 isoform-mediated transcriptional activation of Bcl-xL and Bcl-2 by Ex4a(+)WT1 isoform is likely to be competition with major WT1 isoform for interacting together with the transcriptional co-activators in addition to WT1 itself, ensuing in the abrogation of important WT1 isoform-mediated transcriptional exercise in leukemia cells. Nonethel
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