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Fluenza infection are incompletely resolved. One study concluded that induction of the inflammasome pathway is required for maximal production of antiviral serum IgM and IgG and intranasal IgA (64), while studies in IL-1R-/- mice revealed a distinct deficit in influenza-specific IgM but not IgG or IgA secretion following influenza infection ((65) and Waffarn Baumgarth, unpubl.). Others failed to
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